EXACERBATION OF SYSTOLIC HEART FAILURE QUESTION ANSWER 2022

Running head: EXACERBATION OF SYSTOLIC HEART FAILURE
EXACERBATION OF SYSTOLIC HEART FAILURE
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EXACERBATION OF SYSTOLIC HEART FAILURE1
Table of Contents
Question 1........................................................................................................................................2
Question 2........................................................................................................................................3
Question 3........................................................................................................................................5
Reference.........................................................................................................................................6

EXACERBATION OF SYSTOLIC HEART FAILURE2
Question 1
The patient of the given case study was admitted to the emergency department with the
symptoms of severe dyspnea. The respiratory rate of the patient was 24 breathes/minute which
was quite greater than the normal value of 20 breathes/minute. The concentration of oxygen
saturation level is 85% on the room air where it should be 95% on the room air. The patient had
showed high blood pressure (170/95 mmHg) whereas normally it should be 120/80 mmHg. The
rate of heart beat of the patient had also increased to 120 beats/ minute whereas the normal value
is 70 beats/minute and the auscultation of lungs indicated bilateral basal cracks. The ECG report
showed arterial fibrillation. The patient was identified with severe exacerbation of chronic
systolic heart failure. The chronic systolic failure of heart is a clinical disease in which the
pathological injury or the stress is related with the failure of heart enactment to fulfill the
metabolic demands of the body and thus results in clinical signs. At normal condition the cardiac
output of a person gets increased up to four times during exercise but when a person suffers from
chronic heart failure the person lose this ability. Several intrinsic and extrinsic control
mechanisms are there which control the performance of the heart (Ponikowski et al., 2016). CHF
is considered by the increase in the portfolio of compensatory mechanism involving the
stimulation of renin-angiotensin-aldosterone and sympathetic nervous systems. Maladaptive
compensatory mechanism is initiated which instigate a vicious cycle of advanced myocardial
damage and thus the clinical syndrome gets deteriorated. The pathophysiology of the chronic
systolic heart pressure is determined when the circulatory system gets failed in delivering oxygen
for meeting the metabolic requirements of the body. The mechanism is explained by a critical
interplay in between the intrinsic abnormalities of the function of the ventricular pump and the
external factors which affect the cardiac activity which limits the utilization of oxygen in the