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Clinical Neuropsychology Testing | Report

   

Added on  2020-02-19

14 Pages3098 Words83 Views
Running head: CLINICAL NEUROPSYCHOLOGYName of the studentUniversity nameAuthor’s note

1CLINICAL NEUROPSYCHOLOGYTable of ContentsIntroduction......................................................................................................................................2Presentation of the case history.......................................................................................................2Assessment of results.......................................................................................................................4Differential diagnosis.......................................................................................................................6Additional investigation (refer to the appendix).............................................................................7Intervention plan..............................................................................................................................8Conclusion.......................................................................................................................................9References......................................................................................................................................11Appendix........................................................................................................................................14

2CLINICAL NEUROPSYCHOLOGYIntroduction Neuropsychological testing is a vast area covering the complexities associated withcognition and processing of information. The unravelling and the execution of the informationgoes through a number of mental layers. The fully processed information is then executed into anumber of desirable actions. Thus, neuropsychology relates the functioning of the brain withcognition, emotion and behaviour of an individual. The nerves can be defined as a set of wireswhich forms an entangling network within the brain and is responsible for transforming anddecoding the packets of information into useful behaviour. Evidences and studies have pointed at chemical imbalances along with epigeneticmechanisms to be responsible for the malfunctioning of the brain. The report here presents adetailed analysis of the neuropsychological complexities associated with a patient suffering fromsupranuclear palsy. The case study has been further evaluated with the help of differentialdiagnostic tools and mechanisms. The report presents a well investigated intervention plan formanaging the challenges and the adversities associated with the diseasePresentation of the case history In the present case study the patient had been seen to be suffering from ProgressiveSupranuclear Palsy (PSP). The condition could be described as a gradual degeneration of themajor areas of the brain resulting in loss of cognition and analytical skills in a person. Thedisease could not be related to specific community, race or geography and is equally present bothin the males and the females. As commented by Parsons et al. (2017), no strong genetic pre-disposition or link could be found for development of PSP. Reported study and analysis have

3CLINICAL NEUROPSYCHOLOGYfound a variant in the gene for Tau protein called the H1 Haplotype, which is present onchromosome number 17 and had been have linked with PSP. However, as argued by Sweetetal. (2015), the presence of environmental factors and stress can also result in the accumulation ofthe Tau protein in the brain. The Tau protein is accumulated in an insoluble form inhyperphosphorylated form resulting in neurofibrillary of gliofibrillary tangles. The patient here is a 66 year old Vietnamese right handed lady with ten years of formalschool education and had been diagnosed with progressive Supranuclear Palsy. The patient hadbeen able to perform her daily set of activities independently, though she became relatively slow.The patient has been prescribed Livadopa and Motilium for her current dose of medication. TheLivadopa had been prescribed to her for controlling the development of parkinson’s andParkinson like symptoms. The patient had difficulty in breathing, sleeping and swallowing atnight. The patient had been known to have a history of mild dyspraxia. Dyspraxia ordevelopmental coordination disorder may be classified as lack of sensory perception, spatialawareness and coordination. Further test revealed right frontotemporal cortical abnormalitieswhich made the patient more functional towards the right side. This is because the right side ofthe body is controlled by the left cerebrum and left side by the right cerebrum. As commented byTowns et al. (2017), dyspraxia comes under the specific learning disabilities group affectingthe concentration and the memory of the patient. The patient had shown difficulty in followingthe hand gestures along with poor retropulsion, where the locomotor ability of the person isaffected. Additional co-morbidities such as mild ataxic high blood pressure were also reportedin the patient.

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