Ferguson chest heaviness case PDF

Running head: FERGUSON CHEST HEAVINESS CASE
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Ferguson Chest Heaviness Case
Student’s Name
University

FERGUSON CHEST HEAVINESS CASE
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Ferguson Chest Heaviness Case
Introduction
Breathing challenges are an indicator that the patient is having chest heaviness due to blockage
of arteries which lead to reduced blood circulation thus forcing the heart to struggle with the
limited oxygen in the blood. Patients with such conditions need to learn how to manage the
condition in case the attack appears since in most cases, the patient is required to deal with the
situation immediately to increase the effectiveness of medication and reduce the effects of the
condition.
The pathophysiology of chest heaviness
Mr. Ferguson presents cheat heaviness which is a common symptom for coronary heart artery
disease that blocks arteries thus reducing circulation. The chest heaviness problem is a result of
shortness of breath making it impossible for the patient to breathe well. When cholesterol builds
up in the arteries, it leads to thickening of the walls which make the arterial space narrow thus
limiting blood flow (Tobin, 2010). The outcome is the reduced amount of blood that reaches the
heart muscles which means that the amount of oxygen that the heart receives too is low thus
damaging the endothelium. These effects lead to the accumulation of cholesterol, fats, and
lipoproteins in the artery vessels. Fatty acids are then formed in the artery which produces the
extracellular matrix that forms the atherosclerotic plaque that narrows the luminal
space[ CITATION Car04 \l 1033 ]. The outcome of all this is a narrow cap with an edge that leads to
acute coronary disease. As the plaque deposits grow larger, the blood vessel are narrowed
leading to myocardial infarction which is mostly known as heart attack. This occurs mostly when
the heart fails to function due to insufficient oxygen.

FERGUSON CHEST HEAVINESS CASE
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Pharmacokinetics of the prescribed medications and their indications.
Baren, Brennan, & Brown (2008) suggests that Glyceryl trinate is a common medication for high
blood pressure and heart failure patients by reducing chest heaviness through increasing blood
circulation thus restoring the patient to normal conditions. As a nitrate family drug, it works by
dilating the blood vessels thus increasing blood circulation in the body which restores breathing
to normal thus making the heaviness to disappear. The role of the drug is to reduce heart attacks
that patients with the condition face by restoring the breathing thus reducing the risk of heart
failure in most patients. The drug works by producing nitric oxide which activates for guanlyl
cyclace that leads to the formation of cyclic guanosine monophosphate a compound that is
mostly used to produce nitrogen which makes the arteries and muscles relaxed thus increasing
blood circulation. The mechanism that the drug uses reduce chest heaviness is the fact that
Glyceryl is rapidly absorbed in buccal and sublingual mucosa when inhaled or taken through any
other means thus the effects of the drug can be realized within five minutes of taking. Patients
who do not react to the drug like Ferguson need to visit the nearest health facility or call a doctor
immediately.
For the drug to be effective Kaski, Arrebola-Moreno, & Dungu (2011) suggest that it has to be
taken within a short time after the attack starts so that the reverse mechanism of the drug can be
realized. In most cases, patients who have taken the drug immediately after they sense signs of
an attack have reported positive results. According to Albrecht (2013), the medication becomes
less effective as the patient uses it with time which may be one of the reasons why Ferguson has
not noticed any changes in the disease even after taking several puffs of the spray. The immune
system of the body develops tolerance making the effectiveness of the drug limited from three