This document discusses the health assessment of an elderly woman after swimming and the potential cardiovascular issues she may be facing. It explores the symptoms, pathophysiology, and diagnostic tests for proper management.
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Running head: HEALTH ASSESSMENT HEALTH ASSESSMENT Name of Student Name of University Author note
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1HEALTH ASSESSMENT When Roseanne is an elderly woman of 63 years who returns after swimming – her vitals were found to be much distorted from normal. Firstly, for physical signs showed signs of hypothermia for she had been constantly shivering and her hands were all wrinkled and red. On assessment, her body temperature was found to be at 35 degree which happens to below the physiological normal temperature by 2 degrees. She exhibited signs of tachycardia (102 beats), her respiratory rate is on the higher side (24 breaths/ minute) showing signs of tachypnea and blood pressure is hypertensive (150/84). She is having systolic hypertension. Moreover, given her age and living in loneliness for her husband has died 6 months ago might have worsened her anxiety, negative emotions and self-belief which is quite normal after losing a spouse at this kind of age. These anxieties, sadness and isolation can lead to aggravation of the patient symptoms. Psychological stress and anxiety can narrow down the arteries through a physiological mechanism lead to worsening of hypertension and related cardiovascular dysfunction. Swimming has shown to improve cardiovascular conditions and has been an important regime in cardio vascular rehabilitation programs but care has to be taken, not to irritate or inhibit the respiratory function. Rosseane’s swimming session here might have triggered a cardiac stress which needs to assessed further and managed likewise. The readings as measured before and after the swimming session, differs by a huge margin in terms of blood pressure, heart rate and respiratory rate. A part of it is due to a normal physiological stress induced by bodily exertion during swimming. But given the windy condition and chilly atmospheric condition plus a exertion activity against the rush of water might have triggered something some dire than just a physiological response. The symptoms are persistent and because it is not subsiding like it should – might be indicative of an undiagnosed cardiovascular disorder. At an elderly age, body defenses get weaker and various pathologies set in which can go unnoticed under normal conditions. This is a cue to the pathophysiology underlying the rapid shifts in vital functions parameters beyond the
2HEALTH ASSESSMENT physiologically safe level and as a junior nurse, my first objective is to look for other physical signs like dyspnea, cyanosis, edema, skin flushing (which is present in this case) which can be indicative of a hidden cardiovascular dysfunction. I should be reporting the readings to my mentor as well for an advise on Roseanne’s management. Symptomslikedyspnea,skinflushing,edemaandcyanosiscanbepotential symptoms of a congestive heart condition. In addition, Roseanne exhibits signs of tachycardia which happens as a physiological inotropic effect due to impaired ventricular function. Left ventricular ejection fraction is directly related the pumping action of the heart and production of the stroke volume which becomes a problem in congestive state of heart and left heart failure. This in turn leads to initiation of a physiological tachycardia which again causes the ventricular muscle to expand but without being functional that is - it cannot produce the required stroke volume anymore. Ventricular hypertrophy (Grabneret al.2015) develops as a result of this pathophysiological process and adds to more problems. Decreased stroke volume(George and Steinberg 2015) leads to decrease in the oxygen delivery to the bodily tissues leads to fatigue and decreased lung function. Decreased lung function(Hastiecanet al. 2017)lead to decreased ventilation perfusion ratio and peripheral(Baranoski, Van Leeuwen andChen2017)orevencentralcyanosis(AwasthiandSharma2019)canoccurin cardiovascular system compromised subjects. Hypertension or work related hypertension can be damaging to the whole healing process. Stress induced hypertension(Markouet al.2015) can cause arteries and veins to narrow down through a baroreceptor (Azevedoet al.2016) and chemoreceptor mechanism which lead to an increase in systemic blood pressure and increased preload on the heart – causing more congestion (Mukai 2016) and coronary blood flowdisturbances.Reducedcoronarybloodflowcancauseanginaandotherrelated problems. Moreover lack of blood flow to extremities can reduce thermogenesis and can result in hypothermia, especially in cold temperature conditions and windy conditions.
3HEALTH ASSESSMENT Temperature is regulated by the hypothalamus and these sort of climate can trigger a differential hypothalamic regulation of body temperature in response to chilly external conditions. Body temperature can have a huge impact on the blood circular and how enzymes, organs and tissue functions. Their functions can be readily compromised or inhibited in low bodily temperature and can lead to further constriction of the blood vessels resulting in further and gradual increase in systemic blood pressure beyond the controllable hypertensive level. Shifts in hemodynamics in the body can lead to a development of cardiogenic shock which can be the pathological state presented in Roseanne. Various types of pharmacological and non-pharmacologic interventions has to used imperatively as an integral part of nursing intervention but before that, correct diagnosis of Roseanne health status has to be done to plan the management in an optimized way. As because, the symptoms presents after the swimming exercise and it is definite that the tachypnea, tachycardia and hypertension is exercise induced and vastly differs from the readings when Roseanne is at rest. Hence, a nursing decision has to be taken whether swimming sessions should be allowed for the concerned subject and what does activities should be stopped immediately so as not to aggravate the present cardiovascular conditions of the patient. The cues must be reasoned correctly with clinical knowledge and a patient centered nursing care plan has to be formulated. Further tests like echocardiography , electrocardiography, tread mill test and even holster monitoring based on the degree of cardiovascular severity – has to done as a important part of diagnostic regime to confirm the cues and pathophysiology discussed in the abovediscussion.Moreover,aDopplertestwouldbeimperativeindiagnosingthe functionality of left ventricle with respect to left ventricular ejection fraction. The walls of the ventricles should be checked for any thickening and any occlusion in the coronary artery can be diagnosed with angiography. In addition to this, a daily nursing physical assessment so as
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4HEALTH ASSESSMENT to find any abnormal heart sound or associated abnormal respiratory sound can be suggestive ofaseverecardiorespiratoryconditionandbothacardiovascularandpulmonary management would be required.
5HEALTH ASSESSMENT References : Awasthy, N. and Sharma, R., 2019. Congenitally corrected transposition with isolated atrial septal defect and apicocaval juxtaposition: a hitherto undescribed cardiac cause of central cyanosis.Indian Journal of Thoracic and Cardiovascular Surgery, pp.1-5. Azevedo, L.F., Perlingeiro, P., Hachul, D.T., Gomes-Santos, I.L., Tsutsui, J.M., Negrao, C.E. and De Matos, L.D., 2016. Predominance of Intrinsic Mechanism of Resting Heart Rate Control and Preserved Baroreflex Sensitivity in Professional Cyclists after Competitive Training.PloS one,11(1), p.e0148036. Baranoski, G.V., Van Leeuwen, S.R. and Chen, T.F., 2017, July. Elucidating the biophysical processes responsible for the chromatic attributes of peripheral cyanosis. In2017 39th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC)(pp. 90-95). IEEE. George, P.M. and Steinberg, G.K., 2015. Novel stroke therapeutics: unraveling stroke pathophysiology and its impact on clinical treatments.Neuron,87(2), pp.297-309. Grabner, A., Amaral, A.P., Schramm, K., Singh, S., Sloan, A., Yanucil, C., Li, J., Shehadeh, L.A., Hare, J.M., David, V. and Martin, A., 2015. Activation of cardiac fibroblast growth factor receptor 4 causes left ventricular hypertrophy.Cell metabolism,22(6), pp.1020-1032. Hastie, A., Mauger, D., Denlinger, L.C., Israel, E., Coverstone, A., Fahy, J.V., Phillips, B., Meyers, D.A. and Bleecker, E., 2017. Sputum Eosinophil Longitudinal Variability Effects On Lung Function In The Nhlbi Severe Asthma Research Program (sarp) Cohort.InB33.TH2ENDOTYPEINASTHMA(pp.A3214-A3214).American Thoracic Society.
6HEALTH ASSESSMENT Markou, A., Sertedaki, A., Kaltsas, G., Androulakis, I.I., Marakaki, C., Pappa, T., Gouli, A., Papanastasiou, L., Fountoulakis, S., Zacharoulis, A. and Karavidas, A., 2015. Stress- induced aldosterone hyper-secretion in a substantial subset of patients with essential hypertension.The Journal of Clinical Endocrinology & Metabolism,100(8), pp.2857- 2864. Mukai, Y., 2016. TCTAP C-121 Septal Artery Perforation Occurring During the Wire AdvancementintotheRightVentricle.JournaloftheAmericanCollegeof Cardiology,67(16 Supplement), pp.S237-S239.