The Concept Map - Intracerebral Hemorrhagic Stroke

Running head: INTRACEREBRAL HAEMORRHAGIC STROKE 1
Intracerebral Haemorrhagic Stroke (ICH)
Student’s Name
(997 words)

INTRACEREBRAL HAEMORRHAGIC STROKE 2
The Concept Map - Intracerebral Hemorrhagic Stroke
Symptoms of Hemorrhagic Stroke - Case Study
- loss of speech or difficulty in understanding speech – Karl slurs his words
- confusion and or loss of alertness - appears sleepy, and becomes confused
- nausea and vomiting - Karl vomits
- impairment of consciousness - his pupils cannot turn to the right side
- stiffness or pain in the neck area - his face begins to droop on the left side
- difficulty swallowing - he suddenly has trouble swallowing
The Risk Factors - Intracerebral Hemorrhage
- Hypertension
- Cerebral Cavernous Malfunctions
- Arteriovenous Malformations (AVMs)
Case Study Risk Factors
- Karl has a long history of hypertension
- Age factor (65 years old)
Diagnosis
- A thorough medical and physical background
- Computed tomography (CT) scans
- Magnetic resonance imaging (MRI)
- Blood tests
- MRI angiography
- An electroencephalogram (EEG) or lumbar puncture (spinal tap)
Pathophysiology of the disease the
patient’s clinical manifestations
- Blood flow into his brain
causes severe headache
leading to the below clinical
manifestations:
1. loss of speech or
difficulty in
understanding speech
as he slurs his words
2. confusion and or loss of
alertness as he appears
sleepy, and becomes
confused
3. feels nausea and
vomits
4. has impaired
consciousness as his
pupils cannot turn to
the right side
Intracerebral Hemorrhagic
Stroke – Kennedy Karl
55 years old
Aetiology and the
pathophysiology
- Karl has hypertension
- Bleeding happens inside
his brain
- Blood from an
intracerebral discharge to
form as a mass in the
cerebrum tissues, causing
intraventricular discharge
- Blood may cause intense
hydrocephalus if the drain
breaks into the ventricular
framework
Treatment
- Controlling the bleeding
and reducing mental
pressure
- Drugs that help
counteract effects of
other drugs used by
patients
- Drugs that reduce
hypertension
- Surgery to repair
arteries
- Rehabilitation to
achieve independent

INTRACEREBRAL HAEMORRHAGIC STROKE 3
Risk Factors, Aetiology and Pathophysiology of the Disorder
The common cause of intracerebral hemorrhage (ICH) is protracted arterial high-blood
pressure, which is usually large, single as well as catastrophic. Other risk aspects comprise
cigarette smoking, obesity, excessive alcohol usage; use of cocaine, as well as vessel abnormality
and a high-risk-diet (An, Kim, & Yoon, 2017). People with high blood pressure and above 55
years old are vulnerable to the disorder. The basic underlying driver for the disease is an
aneurysm, a condition that happens when an area of a vein gets amplified from ceaseless and
hazardously hypertension. According to the case study, Karl Kennedy is 65 years of age and has
a history of hypertension, implying that he is vulnerable.
In the case study, his wife notices that his face begins to droop on the left side, and this
sign is a possible indication that the patient is having stiffness or pain in his neck area, and that
makes it impossible or hard for him to turn his face to the right side. The patient appears sleepy
and this signals a state of unconsciousness. Kennedy vomits and becomes confused and these
signs correspond to the intracerebral hemorrhagic stroke’s symptoms of nausea and vomiting.
The pathophysiology involves the functional developments related to the disease. Blood
from an intracerebral discharge aggregates as a mass that can analyze through and pack nearby
cerebrum tissues, causing neuronal brokenness. Blood may cause intense hydrocephalus if the
drain breaks into the ventricular framework - intraventricular discharge (Dadlani & Agrawal,
2017). The patient also demonstrates the impairment of consciousness when it is reported that his
pupils cannot turn to the right side.
How Disorder’s Pathophysiology Manifests
The mass accumulation of blood from the intracerebral hemorrhage dissects through and
compresses the adjacent brain tissues of Kennedy, prompting a neuronal dysfunction.