Skin Cancer Pathology DNA Report 2022

Running Head: SKIN CANCER PATHOLOGY
SKIN CANCER PATHOLOGY
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SKIN CANCER PATHOLOGY 2
Introduction
The body is made up of many cells. The cells develop, rift into fresh cells, and finally,
they die in a delimited scheme. During the young age, the cells divide faster to give room for
growth of the body. During adult life, the cells are no longer dividing faster. In adult life, the
cells divide when they want to swap the ones which are whacked or vanishing to overhaul
injuries. Growth and demise of cells are controlled by the DNA, which exists in the nucleus of
the cells. The DNA offers a plan that gives guidance to the cell in each of the tasks.
The onset of cancer is when the body cells grow in a disorderly manner. The DNA can
repair itself in several mechanisms after being damaged. If the overhaul does not work, the cell
exterminates itself by the method of apoptosis (Guy, Machlin, Ekwueme, & Yabroff, 2015).
In some occasions, neither of these progressions function and therefore, the cad cell carries on
and multiplies short of demise. The old cell passes its defective DNA to the newly formed cells,
and then cancer starts invading tissues. Cancer cells invade the body tissues, something which a
normal cell cannot do (Perera , Gnaneswaran, Staines, & Win, 2015).
There has been an upsurge in the incidence of skin cancer in the recent past. Skin cancer
is the second most prevalent disease in adolescents and young adults. Investigation confirms that
Australia has the uppermost frequency of skin cancer in the world. In 2016, estimates showed
that there were 13, 200 cases of cancer diagnosed in Australia (Apalla, Lallas, Sotiriou, &
Lazaridou, 2017). One thousand seven hundred seventy people were also reported to have died
from cancer in the same year in the country. The rate of mortality stood at an estimated 6.2
deaths per every 100,000 people. Skin cancers develop due to the growth of abnormal cells that
invade the other parts of the body. There exist three primary types of skin cancer which include

SKIN CANCER PATHOLOGY 3
basal cell skin cancer, melanoma, and squamous cell skin cancer (Doran, et al., 2015). The
basal skin cancer and squamous cell skin cancer are commonly known as nonmelanoma skin
cancer. The basal cell skin cancer develops gradually and can harm the tissues that surround it
and can easily lead to death. It occurs as a hard lump that has a scaly top, and in most cases, it
also forms an ulcer (Astell-Burt, Feng, & Kolt, 2014).
Exposure to the violet sun rays increases the chances of getting any of these three types
of cancer. The vulnerability has been growing as a result of the ozone layer. The tanning beds are
also other common causes of skin cancer (Climstein, Furness, Hing, & Walsh, 2016). Basal
cell cancer and melanomas exposure during childhood are particularly harmful. The people who
have light skin are at increased risk of getting skin cancer. The melanoma and squamous-skin
cancer can be reduced by decreasing the exposure to sun rays. It is not known if sunscreen can
affect the risk of basal cell cancer. Nonmelanoma cancer is curable, and treatment involves a
combination of surgery, radiation therapy, and targeted therapy. For the people whose disease
has spread to the entire body, they need quality care so that their life quality is improved. Skin
cancer is the most common type of cancer and accounts for 40% in the world (Roger,
Weinstock, & Feldman, 2015).
Visibly, the tumor is regularly raised, fungating, or might be ulcerated with unpredictable
outskirts. Minutely, tumor cells demolish the cellar layer and structure sheets or smaller masses
which attack the connective tissue. In very much separated carcinomas, tumor cells are
pleomorphic yet looking like typical keratinocytes (Grange, et al., 2015).