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Stroke Risk Factors and Prevention

   

Added on  2020-03-07

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Running head: CLINICAL ASSESSMENT OF TIA LEADING TO STROKEClinical assessment of TIA leading to stroke Name of the StudentName of the UniversityAuthor Note
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1CLINICAL ASSESSMENT OF TIA LEADING TO STROKEExecutive Summary:Stroke happens to be one of the common causes of disability and can lead to death if nottreated on time. Recognising Transient ischemic attack (TIA) in the early stage can preventsubsequent stroke. The specific treatment for preventing stroke is based on the underlyingcause of TIA with cerebrovascular localization followed by other clinical problems.Modifying the risk factors associated with stroke can be done by therapeutic approaches.Therefore by understanding the clinical symptoms of TIA can help in early detection ofstroke and likewise preventive measures can be implemented. The proper maintenance of therisk factors such as controlling blood pressure with controlled cholesterol level, inhibitingsmoking tendency and controlled diabetes can reduce the prevalence of stroke followed byrecurrent TIA. In this report the management of TIA in primary care through properassessment, diagnosis followed by preventive measures to reduce the occurrence of strokehas been discussed with respect to a patient diagnosed with TIA.
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2CLINICAL ASSESSMENT OF TIA LEADING TO STROKETable of Contents1. Patient situation:.....................................................................................................................31.1. Previous history:..............................................................................................................32. Primary assessment:...............................................................................................................32.1. Information gathered:......................................................................................................32.2. Current situation:.............................................................................................................42.3. Epidemiology with aetiology:.........................................................................................42.4. Undertaking the diagnosis:..............................................................................................52.5. TIA symptoms:................................................................................................................53. Investigations:........................................................................................................................63.1. Management of the situation:..........................................................................................73.2. Prognosis:........................................................................................................................83.3. Preventing future occurrence:.........................................................................................84. References:.............................................................................................................................9
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3CLINICAL ASSESSMENT OF TIA LEADING TO STROKE1. Patient situation: A 59 year old widow named Mrs. Amari was brought to the emergency department byher son, Niko. He noticed a sudden slurring in her mother’s speech with drooped face on oneside. She reported her son about the numbness of right side of her face including her rightarm. Niko brought her to the hospital after having a fear of stroke.1.1. Previous history: Her mother had a history of hypertension with hypercholesterolemia with 25 years oftobacco usage which she had stopped ten years ago. She showed a positive history of familyline with heart disease. She does not follow a regular exercise regimen with occasional walkin neighbourhood. 2. Primary assessment:The nursing staff in the emergency department assessed her and found a slightdiversion of mouth in the right side with speech slightly slurred. Further assessment showedno such weakness with steady gait and could swallow without any difficulty. She followedall the commands and looked oriented. The pupal examination showed round, equal andnormally reacting to light (4mm to 2mm). No such nystagmus was noted. She did notcomplain of any kind of headache followed by nausea, vomiting, chest pain, diaphoresis orvisual complaints.2.1. Information gathered: On examining her vital signs her temperature was 36.7ºC with an increased bloodpressure of 148/97mmHg, pulse recorded 81, respiratory rate was 14 and the oxygensaturation was 94%. Numbering through Glasgow Coma Scale showed 15 with 6.6mmol/Lblood glucose level. Based on the history and examination result she was diagnosed by a
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